PSYCH OpenIR  > 健康与遗传心理学研究室
The functional role of peroxiredoxin 3 in reactive oxygen species, apoptosis, and chemoresistance of cancer cells
Li, Lianqin1; Yu, Ai-Qun2,3,4
2015-12-01
Source PublicationJOURNAL OF CANCER RESEARCH AND CLINICAL ONCOLOGY
ISSN0171-5216
SubtypeReview
Volume141Issue:12Pages:2071-2077
AbstractThe mammalian peroxiredoxin (PRX) family contains six members that provide antioxidant defense in different cell types by removing reactive oxygen species (ROS) through conserved active cysteines. Different from other members, PRX3 is predominantly located in mitochondria, a major apoptosis mediator. The purpose of this review is to summarize the findings on PRX3 concerning its role in ROS removal, apoptosis, and chemoresistance of cancer cells.
KeywordPeroxiredoxin (PRX) Reactive oxygen species (ROS) Oxidative stress Apoptosis Cancer
Indexed BySCI
Language英语
WOS IDWOS:000364224800001
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Cited Times:12[WOS]   [WOS Record]     [Related Records in WOS]
Document Type期刊论文
Identifierhttp://ir.psych.ac.cn/handle/311026/15311
Collection健康与遗传心理学研究室
Affiliation1.Binzhou Med Univ, Dept Obstet & Gynecol, Yantai Affiliated Hosp, Yantai 264100, Peoples R China
2.Chinese Acad Sci, Inst Psychol, Beijing 100101, Peoples R China
3.Chinese Acad Sci, Grad Sch, Beijing 100049, Peoples R China
4.Beijing Technol & Business Univ, Canvard Coll, Beijing 101118, Peoples R China
Recommended Citation
GB/T 7714
Li, Lianqin,Yu, Ai-Qun. The functional role of peroxiredoxin 3 in reactive oxygen species, apoptosis, and chemoresistance of cancer cells[J]. JOURNAL OF CANCER RESEARCH AND CLINICAL ONCOLOGY,2015,141(12):2071-2077.
APA Li, Lianqin,&Yu, Ai-Qun.(2015).The functional role of peroxiredoxin 3 in reactive oxygen species, apoptosis, and chemoresistance of cancer cells.JOURNAL OF CANCER RESEARCH AND CLINICAL ONCOLOGY,141(12),2071-2077.
MLA Li, Lianqin,et al."The functional role of peroxiredoxin 3 in reactive oxygen species, apoptosis, and chemoresistance of cancer cells".JOURNAL OF CANCER RESEARCH AND CLINICAL ONCOLOGY 141.12(2015):2071-2077.
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